Visceral Manipulation in Osteopathy

I Foundations and Techniques

1 Visceral Manipulation according to Barral

2 Fascial Treatment of the Organs according to Finet and Williame

3 Circulatory Techniques according to Kuchera

4 Reflex Point Treatment according to Chapman

1 Visceral Manipulation according to Barral

Theory of Visceral Manipulation

Physiology of Organ Movement

We distinguish three movements of the internal organs: motricity, mobility, and motility.

Motricity

Motricity refers to passive changes in the position of the organs that result from arbitrary motor activity by the locomotor system.

If, for example, you bend the upper body to the right, the move compresses the abdominal organs on the right side but stretches the wall of the torso on the left, resulting in a pull on the left-sided organ attachments which enlarges their available space.

When bending the upper body forward, the intraperitoneal organs migrate anteriorly as the result of gravity and their high degree of mobility.

Any activity that involves continuous sitting compresses the small and large intestines and impairs their peristalsis.

Lifting both arms in maximum flexion results in an extension of the thoracic spinal column (TSC) and an inspiration position of the ribs. As the parietal pleura follows this movement of the thorax, and the lung is connected to the movement of the chest by its stretch, the lung increases its volume without having to make any additional respiratory effort.

Mobility

In visceral manipulation, mobility refers to the movement either between two organs or between an organ and the wall of the torso, the diaphragm, or another structure in the musculoskeletal system. The engine for this movement can be motricity or different "automatisms."

Automatism refers to a movement that is performed involuntarily by striated or smooth muscles. Furthermore we can differentiate between automatisms that occur continuously and movements of the organs marked by periodicity.

Automatisms include:

diaphragmatic breathing

heart action

peristalsis of the visceral hollow organs in the gastrointestinal tract

Diaphragmatic breathing. With 12-14 breaths/min, the diaphragm contracts about 20000 times a day. In doing so, it acts like a piston sliding up and down in a cylinder. During inspiration, the diaphragm sinks caudally, the volume of the thorax increases, and the abdominal organs migrate downward. The soft muscular abdominal wall allows the abdominal organs to move anteriorly out of the way; as a result, the volume of the abdomen hardly changes at all during inspiration.

During expiration, the opposite movement occurs.

Heart action. At 70 heartbeats/min, the heart contracts about 100000 times a day. These actions act like vibrations on the mediastinal organs and, via the diaphragm, also on the abdomen.

Motility

Motility is defined as the intrinsic movement of the organs with a slow frequency and small amplitude. It can be detected by the hand of a trained practitioner and is the kinetic expression of movements in the organ tissues. During embryonic development, the evolving organs carry out growth movements and position shifts that remain stored in each organ cell as a kind of memory. Motility is a rhythmic repetition of this embryonic migration to its place of origin and back to the final, postnatal position.

Likewise, it is impossible to rule out a connection to the craniosacral rhythm, in spite of the fact that motility shows a different frequency.

We distinguish between a so-called expiration phase, that is, the movement toward the median line, and an inspiration phase, a movement in the opposite direction away from the median line.

The frequency is 7-8 cycles/min, one cycle comprising one expiration and one inspiration.

Visceral Joint

Motricity, automatisms, and mobility cause changes in positional relationships of the organs. The movement occurs along a defined axis with a defined amplitude and, thereby, the organs with structural relationships to each other act in a similar way to a joint in the locomotor system.

Two joint partners form the visceral joint; the two joint partners can be two organs (liver-kidney) or an organ and a muscular wall (liver-diaphragm).

The joint partners have surfaces that glide toward each other; the visceral joint partners are separated from each other by a capillary gap, and the surface of their gliding face is smooth and covered with a film of fluid.

The serous membranes-pleura, peritoneum, pericardium, and meninges/peripheral nerve sheaths-constitute most of these gliding surfaces.

The joint partners are fixed to each other: there are several attachments on the organs that are important for the axis of movement-see box.

Note

Organs are attached by:

• The double-leaf system
• The ligamentary system
• Turgor and intracavitary pressure
• The mesenteries
• The omenta

Double-Leaf System

Wherever we find a film of fluid (peritoneum, pleura, pericardium), the organs of a visceral joint are both separated from each other and connected by this fluid. They act in a similar way to two panes of glass with a drop of fluid between them-they can glide past each other, but the adhesive force keeps them together.

Ligamentary System

In visceral manipulation, ligaments are pleural or peritoneal folds that connect an organ to either the wall of the trunk or other organs. In most cases, they do not contain blood vessels but are sensitive and well innervated. They fix the organs against gravity.

Turgor and Intracavitary Pressure

Turgor or intravisceral pressure refers to the ability of an organ to occupy the largest space possible. The reasons for this characteristic are elasticity, vascular effects (decreased or increased blood circulation), and gases in hollow organs.

The intracavitary pressure is the sum of all intravis-ceral pressures plus the pressure between the organs.

This pressure causes the organs to be pressed and fixed against each other. As a result, we find a large excess of pressure in the abdomen, which is countered by a vacuum in the thorax. The diaphragm is the border layer between these pressure states. The organs near the diaphragm are influenced greatly by pressures. A diaphragmatic hernia will thus always lead to a movement of organ parts from the abdomen into the thorax, against gravity. This illustrates the great potency of such pressure effects on the fixation of the organs.

Mesenteries

The mesenteries are duplicatures the peritoneum with only a minor role in fixation. They supply the organ's blood circulation.

Omenta

The omenta are also infoldings of the peritoneum that connect two organs to each other. Their role in organ fixation is rather small, although their vasculonervous function is of more importance.

Pathology of Organ Movement

Organs move around specific axes and with defined amplitudes. Changes in the axes of movement or amplitudes lead to deviations from the physiologic mobility or motility. Such changes lead to

local pathologies first without and later with symptoms

recurring local pathologies

pathologies in visceral or parietal regions of the body that are linked via topographic, vascular, nervous, or fascial osteopathic chains

In principle, we distinguish between disturbed mobility and disturbed motility.

Disturbed Mobility

An organ completely or partly loses its ability to move as a result of the following causes.

Note

Causes of disturbed mobility:

• Adhesions/fixations
• Viscerospasm
• Ptosis

Articular restrictions. This dysfunction can lead to disturbed mobility and disturbed motility. If only the motility but not the mobility is disturbed, we speak of "adhesions." If, however, both movement qualities are impaired, we call this "fixations."

In fixations, the axis of movement and the amplitude could have changed. Causes include:

infections inflammation

surgical interventions

blunt trauma

Muscular restrictions (viscerospasms). Viscerospasms affect only the hollow organs (e.g., stomach, intestines, or ureters). Irritation of the organ can lead to nonphysiologic contraction of the smooth muscles accompanied by impaired organ functions.

As a result, we notice a change in motility, especially in amplitude. Altered mobility affects the organ only when the viscerospasm has also adversely impacted the organ attachments.

Causes for irritations include:

inflammation

vegetative dysinnervation

allergic reactions

psychosomatic influences

Loss of ligamentary elasticity (ptosis). The loss of elasticity in ligamentary attachments causes diverse organs, such as the transverse colon, kidney, or urinary bladder, to descend with gravity.

The axes and amplitude of mobility change, as does motility, the causes of which include:

a result of adhesions

asthenic constitution

anorexia or rapid weight loss due to other causes

age-related loss of elasticity

depression with generalized tonus reduction

general laxity at the end of or after pregnancy

delivery by vacuum...