Selye pioneered the concept of biologic stress and contributed to the exploration of this field for over 40 years. He established the Hans Selye Foundation in 1980 to promote research and education on stress. In the spirit of the Foundation, Dr. Marc Cantin, late president, and Drs. Sandor Szabo and Yvette Tache, vice presidents, have established the Hans Selye Symposia on Neuroendocrinology stress conference and book series. So far three symposia have been held in Montreal where Selye started to work in the 1930's. These symposia bring together laboratory and clinical investigators engaged in the study of the neuroendocrinology, pathophysiology of stress, and related fields. The first Hans Selye Symposium, held in 1986, was devoted to "Neuropeptides and Stress" and the proceedings were published by Springer Verlag. In 1992, in commemoration of the tenth anniversary of Selye's passing in October 1982, another Hans Selye Symposium was organized and was entitled "Corticotropin-Releasing Factor and Cytokines: Role in the Stress Response". The proceedings recently appeared in 1993 as volume 697 of the New York Academy of Sciences.
A substantial part of the present volume originates from the Hans Selye Symposium and devoted to the "Neuroendocrinology of Gastrointestinal Ulceration".
Reihe
Auflage
Softcover reprint of the original 1st ed. 1995
Sprache
Verlagsort
Zielgruppe
Für Beruf und Forschung
Research
Illustrationen
Maße
Höhe: 244 mm
Breite: 170 mm
Dicke: 15 mm
Gewicht
ISBN-13
978-1-4613-5759-9 (9781461357599)
DOI
10.1007/978-1-4615-1867-9
Schweitzer Klassifikation
Neuroendocrine Factors in Ulcer Pathogenesis.- Role of Sensory Neurons in the Control of Gastric Mucosal Blood Flow and Protection.- Sympatho-Adrenergic Regulation of Duodenal Mucosal Alkaline Secretion.- Potentiation of Intestinal Secretory Responses to Histamine: Pathophysiological Implications.- Acidosis Plays an Important Role in Stress-Induced Gastric Ulceration in Rats.- The Development of Acute Gastric Mucosal Lesions Following Burn Stress.- Experimentally Induced Duodenal Ulcers in Rats Are Associated with a Reduction of Gastric and Duodenal Calcitonin Gene- Related Peptide-Like Immunoreactivity.- The Effect of bFGF and PDGF on Acute Gastric Mucosal Lesions, Chronic Gastritis and Chronic Duodenal Ulcer.- Animal Models and Pathogenesis of Inflammatory Bowel Disease.- Leukotrienes in Human and Experimental Ulcerative Colitis.- The Brain-Gut Axis and the Mucosal Immunoinflammatory Response.- Brain-Gut Interactions in Ulcer Pathogenesis.- Neuroendocrine Control of Gastric Acid Secretion.- Role of Corticotropin-Releasing Factor in Stress-Induced Changes in Gastrointestinal Transit.- Stress, Corticotrophin-Releasing Factor (CRF) and Gastric Function.- Corticotropin Releasing Factor and Gastric Erosions.- The Limbic System and Stress Ulcers.- Neurotensin Acts in the Mesolimbic Dopamine System to Affect Gastric Mucosal Function.- Role of Medullary Trh in Vagally Mediated Formation of Gastric Lesions.- Role of the Vagal Nerve in the Development of Gastric Mucosal Injury and Its Prevention by Atropine, Cimetidine, ?-Carotene and Prostacyclin in Rats.- The Role of Vagal Innervation in Adaptive Cytoprotection.- A Biobehavioral Profile of an Ulcer Susceptible Rat Strain.- Novel Therapeutic Approaches to Gastrointestinal Ulceration.- The Gastric Mucosal Barrier: a Dynamic,Multifunctional System.- New Approaches to Gastroprotection: Calcium Modulators.- Efficacy of Dopaminergic Agents in Peptic Ulcer Healing and Relapse Prevention: Further Indication of the Importance of Stomach Dopamine in the Stress-Organoprotection Concept.- Dietary Factors Influencing Gastrointestinal Ulceration: the Luminal Regulatory System.- Contributors.
