Brain Edema XI
Proceedings of the 11th International Symposium, Newcastle-upon-Tyne, United Kingdom, June 6-10, 1999
Published on 12. June 2002
Book
Paperback/Softback
XIX, 572 pages
978-3-7091-7257-5 (ISBN)
Description
Brain edema is found in a wide variety of clinical disorders including stroke, intracerebral haemorrhage, subarachnoid haemorrhage, head injury, brain tumors and hydrocephalus.
This volume brings together clinical and basic scientists from all over the world. Their expertise in the understanding of brain edema and shifts in brain water compartments has led to a further significant step in our understanding of those diseases characterized by brain edema. This book has also drawn on the expertise of the International Advisory Board of the Brain Edema Society, who have carefully summarized each section, thus providing an easy-to-read summary of the latest advances in each subject. The book is therefore much more than a collection of papers: it represents a critical appraisal and puts each paper into modern scientific context.
The greatest advances have come from the rapid development of modern imaging techniques, especially with magnetic resonance imaging (MRI). Imaging can now produce "water maps" and "metabolic profiles" that bring brain metabolism and water content right into every clinic with access to MRI.
This book provides the background knowledge to understand these pathophysiological changes.
This volume brings together clinical and basic scientists from all over the world. Their expertise in the understanding of brain edema and shifts in brain water compartments has led to a further significant step in our understanding of those diseases characterized by brain edema. This book has also drawn on the expertise of the International Advisory Board of the Brain Edema Society, who have carefully summarized each section, thus providing an easy-to-read summary of the latest advances in each subject. The book is therefore much more than a collection of papers: it represents a critical appraisal and puts each paper into modern scientific context.
The greatest advances have come from the rapid development of modern imaging techniques, especially with magnetic resonance imaging (MRI). Imaging can now produce "water maps" and "metabolic profiles" that bring brain metabolism and water content right into every clinic with access to MRI.
This book provides the background knowledge to understand these pathophysiological changes.
Reviews / Votes
"... Fuer jeden, der den aktuellen Stand der Forschung auf diesem Gebiet einsehen und erfassen will, ist dieser XI. Band 'Brain Edema' dringend zu empfehlen." Rechtsmedizin 5/2003 Band 13, Heft 2More details
Series
Edition
Softcover reprint of the original 1st ed. 2000
Language
English
Place of publication
Vienna
Austria
Publishing group
Springer Wien
Target group
Professional and scholarly
Illustrations
211
10 farbige Abbildungen, 211 s/w Abbildungen
10 Illustrations, color; 211 Illustrations, black and white; XIX, 572 p. 221 illus., 10 illus. in color.
Dimensions
Height: 28 cm
Width: 21.6 cm
ISBN-13
978-3-7091-7257-5 (9783709172575)
DOI
10.1007/978-3-7091-6346-7
Schweitzer Classification
Other editions
Additional editions
A.D. Mendelow | A. Baethmann | Z. Czernicki
Brain Edema XI
Proceedings of the 11th International Symposium, Newcastle-upon-Tyne, United Kingdom, June 6-10, 1999
E-Book
12/2012
Springer
€149.79
Available for download
A.D. Mendelow | A. Baethmann | Z. Czernicki
Brain Edema XI
Proceedings of the 11th International Symposium, Newcastle-upon-Tyne, United Kingdom, June 6-10, 1999
Book
05/2001
Springer
€160.49
Article exhausted; check different version
Content
Imaging: 1H-MR Spectroscopic Monitoring of Posttraumatic Metabolism Following Controlled Cortical Impact Injury: Pilot Study (M. U. Schuhmann, D. Stiller, S. Thomas, T. Brinker, and M. Samii); Heterogeneous Mechanisms of Early Edema Formation in Cerebral Contusion: Diffusion MRI and ADC Mapping Study (T. Kawamata, Y. Katayama, N. Aoyama, and T. Mori); Metabolic Changes in Pericontusional Oedematous Areas in Mild Head Injury Evaluated by 1H MRS (B. C. Son, C.-K. Park, B.-G. Choi, E.-N. Kim, B.-Y. Choe, K.-S. Lee, M.-C. Kim, and J.-K. Kang); Malignancy of Brain Tumors Evaluated by Proton Magnetic Resonance Spectroscopy (1H-MRS) in Vitro (Z. Czernicki, D. Horsztynski, W. Jankowski, P. Grieb, and J. Walecki); rCBF in Hemorrhagic, Non-Hemorrhagic and Mixed Contusions After Severe Head Injury and its Effect on Perilesional Cerebral Blood Flow (B. M. Hoelper, M. M. Reinert, A. Zauner, E. Doppenberg, and R. Bullock).- Molecular Mechanisms: Glutathione Homeostasis and Leukotriene-Induced Permeability in Human Blood-Brain Barrier Endothelial Cells Subjected to in Vitro Ischemia (A. Muruganandam, C. Smith, R. Ball, T. Herring, and D. Stanimirovic); Comparison of NAA Measures by MRS and HPLC (P. P. Fatouros, D. L. Heath, A. Beaumont, F. D. Corwin, S. Signoretti, R. H. AL-Samsam, B. Alessandri, P. Lazzarino, R. Vagnozzi, B. Tavazzi, R. Bullock, and A. Marmarou); Influence of the Bradykinin B1/B2-Receptor-Antagonist B 9428 on the Cerebral Microcirculation and Outcome of Gerbils from Global Cerebral Ischemia (J. Lehmberg, J. Beck, A. Baethmann, and E. Uhl); Evolution of Energy Failure after Repeated Cerebral Ischemia in Gerbils (T. Kuroiwa, U. Ito, Y. Hakamata, S. Hanyu, G. Mies, and D. Hermann); Indomethacin and Cyclosporin a Inhibit in Vitro Ischemia-Induced Expression of ICAM-1 and Chemokines in Human Brain Endothelial Cells (W. Zhang, C. Smith, R. Monette, J. Hutchison, and D. B. Stanimirovic); The Selectin Superfamily: The Role of Selectin Adhesion Molecules in Delayed Cerebral Ischaemia After Aneurysmal Subarachnoid Haemorrhage (J. J. Nissen, D. Mantle, A. Blackburn, J. Barnes, T. Wooldridge, B. Gregson, and A. D. Mendelow); Traumatic Brain Injury in Mice Deficient in Poly-ADP(Ribose) Polymerase: A Preliminary Report (M. J. Whalen, R. S. B. Clark, C. E. Dixon, P. Robichaud, D. W. Marion, V. Vagni, S. Graham, L. Virag, G. Hasko, R. Stachlewitz, C. Szabo, and P. M. Kochanek); Increased Immunolocalization of Nitric Oxide Synthases During Blood-Brain Barrier Breakdown and Cerebral Edema (S. Nag, P. Picard, and D. J. Stewart); Biological Functions of Extravasated Serum IgG in Rat Brain (E. Kadota, Y. Muramatsu, K. Nonaka, M. Karasuno, K. Nishi, K. Dote, and S. Hashimoto); Possible Reverse Transport of ß-Amyloid Peptide Across the Blood-Brain Barrier (R. Pluta, A. Misicka, M. Barcikowska, S. Spisacka, A. W. Lipkowski, and S. Januszewski); Reducing Conditions Produce a Loss of Neuroprotective Efficacy of Competitive but not Non-Competitive Antagonists in a Model of NMDA-Mediated Excitotoxicity in Organotypic Hippocampal Slice Cultures (A. K. Pringle, J. Self, and F. Iannotti); Role of Nitric Oxide in Blood-Brain Barrier Permeability, Brain Edema and Cell Damage Following Hyperthermic Brain Injury. An Experimental Study Using EGB-761 and Gingkolide B Pretreatment in the Rat (H. S. Sharma, K. Drieu, P. Alm, and J. Westman); Extracts of Ginkgo Biloba and Panax Ginseng Protect Brain Proteins from Free Radical Induced Oxidative Damage in Vitro (M. S. Siddique, F. Eddeb, D. Mantle, and A. D. Mendelow); p-Chlorophenylalanine, an Inhibitor of Serotonin Synthesis Reduces Blood-Brain Barrier Permeability, Cerebral Blood Flow, Edema Formation and Cell Injury Following Trauma to the Rat Brain (H. S. Sharma, T. Winkler, E. Stälberg, S. Mohanty, and J. Westman); S-100 Serum Levels and Outcome After Severe Head Injury (R. D. Rothoerl, C. Woertgen, and A. Brawanski); A Microstructural Study of Spinal Cord Edema (H. Naruse, K. Tanaka, and A. Kim);