Developments in Ophthalmology / Cataract Pathogenesis: Results of Epidemiological Studies and Experimental Models
Cataract '93. 4th International Cataract Epidemiology Meeting and 6th Scheimpflug Club Meeting, Melbourne, March 1993
Published in 1994
Book
Hardback
VIII, 100 pages
978-3-8055-5987-4 (ISBN)
Description
Cataract is responsible for the blindness of over 20 million people worldwide. Though operative treatment is possible, the need far outstrips current surgical capacity - less than one in five will be able to have surgery. Why cataract occurs, how frequently it occurs, and how its onset or progress might be checked are among the questions dealt with in this volume. This publication contains papers presented at the 4th International Cataract Epidemiology Meeting held in Melbourne, Australia. The great variety of interesting material included reflects the wide range of interests of clinicians and researchers in this fascinating field. The volume concentrates on cataract pathogenesis and includes current methods for the classification of cataracts, study guidelines and the results of epidemiological studies. It is important reading for ophthalmologists and epidemiologists concerned with cataract.
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Content
A population-based estimate of cataract prevalence - the Melbourne visual impairment project experience, P.M. Livingston et al; selection of populations and samples in the design of epidemiological studies of cataract, C.S. Guest; diabetes, sex and cataract, J.J. Harding and M. Egerton; surgical result of radiation-induced cataract in Chinese patients with nasopharyngeal carcinoma, S.K. Kwok et al; judgement of cataractous changes applying a new system of image analysis in epidemiological studies, K. Sasaki et al; population-based case-control study of cortical cataract in the Noto area, Japan, T. Shibata et al; population-based study of risk factors in senile cataract, K.H. Shyn et al; influence of topical monomethyl trisilanol on cataract epidemiology, Verin et al; lens heating in donor eyes by VHF and UHF radiation, H. Maclean; serum tryptophan and age-related cataract, A.J. Elderfield and R.J.W. Truscott; in vivo assessment of an animal model of diabetic cataract - medical intervention studies, T.L. Joon et al; modelling cortical cataractogenesis 14 - reduction in lens damage in diabetic rats by a dietary regimen combining vitamins C and E and betacarotene, F. Kilic et al; modelling cortical cataractogenesis 15 - use of combined dietary anti-oxidants to reduce cataract risk, J.R. Trevithick et al; tryptophan metabolism and the reactive metabolite hypothesis for human cataract, R.J.W. Truscott and A.M. Wood; lenses with retrodots do not have a high oxalate content, B.S. Lewis et al; distribution pattern of viable mitochondria in bovine lens epithelial cells, O. Leip et al.